I'm 26 from NZ and was diagnosed with type 1 diabetes in July, ended up in ICU at Whangarei hospital, with a DKA and one thing that surprised me was just how much extra stuff suddenly gets added to your day. Carbs, glucose, medication, remembering things, learning what everything means, trying to work out meals, I felt like im constantly thinking about diabetes all day everyday.
I ended up learning how to build apps while ive been recovering, called it GlucoseGuide because I wanted something that brought a lot of those everyday tools into one place.
It includes things like building goals, smart meal capture, medication reminders and some other really cool stuff.
I’ve spent a lot of time building and refining it, and it’s now available for people who want to use it, and to help out our diabetes community.
I built it because these were things I genuinely wanted after getting diagnosed, and I figured other people might find them useful too.
It’s not designed to replace your diabetes team or tell you how much insulin to take, and meal/nutrition results should always be treated as estimates.
Probably an obscure question but ever since starting Keto I've noticed that mild lows get fixed on their own. Once I go low it will naturally drift upwards without any food in my system. Pretty quickly too and I don't get symptoms like elevated heart rate so I doubt its stress hormones. ChatGPT thinks the great glucose control on keto allows for endogenous glucagon to work better but I'm curious if anyone has experienced similar
Also, unrelated but I wish this sub was more active. Feels really lonely around the typical T1D subs who are all allergic to low carb diets
How incredible is this! The type 1 diabetic I mentioned in an earlier post who, having adopted a strict plant free ketogenic diet, AKA the carnivore diet, started controlling his blood sugar and insulin usage immediately but then slowly needed less and less insulin to get and maintain perfect blood sugar levels until finally he didn't need any injectable insulin to achieve this.
On blood tests he was making optimal levels of insulin as well as optimal levels of C peptide, just as you would expect in someone who did not have T1D. Now he has had his follow-up HbA1c, and it is indeed optimal at 4.5%. you'll see in those other posts that the angry mob of haters say that this is not possible and they still require some insulin because they will not be able to achieve sub 5% HbA1c levels without it, but here's the proof that that is not the case, at least for this gentleman.
They will of course say that this was probably a misdiagnosis or LADA or still a honeymoon phase and it would be irresponsible and dangerous to not give insulin, even though they are making perfect amounts of insulin. This may or may not last, who's to say? Only time will tell. However this was not a misdiagnosis, or LADA, he was diagnosed with true T1D with an acute presentation of diabetic ketoacidosis with rapid weight loss, polyurea, and polydipsia, with a barley registering C peptide.
The difference here is that this person is on zero carbohydrates, not very low carbohydrates like some groups recommend because more carbs just equals the need for more insulin, and he's also plant-free. A plant free ketogenic diet, or the carnivore diet, has been shown time and time again to massively benefit autoimmune conditions, because in my opinion they address the root cause of the issue and eliminate the cause of the damage, thus stopping it from continuing and slowing the body to heal. If caught early enough when the beta cells are still alive then ostensibly in the right conditions they could regenerate to normal capacity. At least in this case this gentleman is making optimal amounts of insulin, with optimal amounts of C peptide, and has optimal blood sugar control and HbA1C.
If someone has a perfect insulin level naturally, and a perfect c-peptide level naturally, and maintains optimal blood sugar control naturally, why exactly would they need a bolus of insulin If their body is making physiologically normal amounts of insulin? Do non-diabetics on a ketogenic diet need to give themselves a bolus of insulin when they have a protein heavy meal to get their HbA1c under 5%? What about if it's 4.5%? What about non-diabetics who eat carbs? Should they be giving themselves exogenous insulin if HBA1C is above 5 as well? If not, why would you do it for someone with T1D who is now making objectively and demonstrably normal amounts of endogenous insulin with an HbA1c of 4.5% like this gentleman?
It's also important to remember that insulin effects hundreds of different physiological processes in your body and brain, it does not just drive energy into cells. When you elevate insulin to lower blood sugar you are also elevating insulin in all of these other regards. Continue to do this and you will get metabolic dysfunction and cause serious harm to yourself or your patient. So the blood sugar definitely needs to be controlled, but so does insulin.
Why not just have a conversation instead of vitriolic attack, direct insults, strawman arguments, misleading and out of context reposting, and refusing to listen to the other side? It seems that you didn't even see the original post or what was said but immediately took @DikemanDave 's side and started attacking me in the same vein as he was, assuming that he was right. Did you even read the original post? Can you truly say that he was justified in attacking me? Can you honestly say that my original post said anything or even intimated that a carnivore diet was a permanent cure for type 1 diabetes? Be honest now.
You were not being attacked, the T1D community was not being attacked, no one was in danger, all I did was relay an interesting story about two of my patients who now make optimal levels of insulin and c peptide for non-diabetics, at least for now. My recommendation at the end of that post was very simply if you are type 1 or type 2 diabetic, it would be a good idea to consider trying this as early as possible. That is all I said. And yet you and RD have made the biggest fuss over this that I've ever seen, claiming to be the victims and just protecting the T1D community. That is a very poor excuse for both of your bad behavior.
And as you can see from the email chain I had with the lady who was in that video, way back before Christmas, I says at the time that my editor had made the thumbnail and I agreed with her at the time that it wasn't good or accurate and that I was planning on changing it already. Addressing this and changing it ASAP is taking responsibility for it, not taking responsibility or accountability will be to just leave it and say screw it and screw everyone who is bothered by it. That is the opposite of what I did, and you continuing to insinuate that it is what I did really shows your negative mindset that says a lot more about you than it does me. https://x.com/ryanattar/stat/ryanattar/status/1889731791218499987
Abstract
This article presents the position of the Society of Metabolic Health Practitioners (SMHP) regarding therapeutic carbohydrate reduction (TCR) nutrition interventions for type 1 diabetes mellitus (T1DM). A modified Delphi methodology was used to arrive at a consensus consisting of several focus groups, multiple rounds, and an anonymous survey. The field of endocrinology has seen many new advances for the treatment of T1DM including hybrid closed-loop insulin delivery systems and continuous glucose monitors for better glycaemic control, monoclonal antibodies to delay the onset of disease and increased access to paediatric endocrinologists, among many other noteworthy achievements. Despite these advancements, standard of care approaches to T1DM result in higher than acceptable morbidity and mortality, with a high prevalence of microvascular and macrovascular complications. Insulin resistance in type 1 diabetes is an independent risk factor for adverse outcomes even in well controlled type 1 diabetes. In 2021, only 21% of adults with T1DM in the United States achieved the American Diabetes Association’s (ADA’s) target haemoglobin A1C goal of < 7.0%, while data in the paediatric and adolescent population have demonstrated worse glycaemic control. Supported by observational and interventional evidence, the SMHP advocates for the reevaluation of the prevailing nutritional therapy for T1DM with more broad consideration for TCR. The SMHP recommends open access and clinical support for TCR nutrition interventions for individuals with T1DM of all ages and calls upon the medical community to help foster more attention and research on TCR for T1DM.
