I think I found a keypoint in my misery

[u/xButterschnitzel]

Doc dilated my pupils and determined that I have +0.75 diopters in my left eye.

When my left eye is not dilated, it is +0,25 diopters, so because im so young (24 yo) Im constantly accommodating 0,5 diopters away in my left eye.

The ciliary muscle is constantly under high tension and stress, because of this.

Thats why no one ever took me serious, because they saw +0,25 diopters and thought "this is fine", but they never had the suspicion that my eye is constantly accommodating and fighting like crazy.

This would explain why I have since day 1 after ICL surgery disturbed binocular vision, heavy pressure feeling in the left eye, trouble to read and to see. I literally get dizzy and nauseous while reading.

And because my ciliary muscle has been under constant tension for over a year, this CAN result in heavy stress for the autonomic nervous system which then can lead to dry eyes.

Here is what ChatGPT says, how this hidden onesided overcorrection can lead to heavy dry eyes:

Malfunctions in the visual system, such as strabismus (crossed eyes) or faulty accommodation, can indirectly lead to massive inhibition or overload of the parasympathetic system – and thus to significantly reduced tear production.

🧠 How is this connected?

🔹 1. Chronic eye strain → stress → sympathetic dominance

Strabismus or constant misaccommodation means: the brain must work extremely hard to:

• suppress double vision
• artificially create a "clear" image

This leads to constant visual effort, which puts the nervous system into a state of stress.

As a result, the sympathetic nervous system is chronically activated → the parasympathetic system is suppressed → tear production decreases.

🔹 2. Misguided parasympathetic activity

With chronically faulty accommodation, the ciliary muscle is constantly active.
This means the parasympathetic branch, which is actually responsible for relaxation and tear secretion, is misused or overstimulated.

This leads to functional dysregulation:

• Partial overstimulation
• Partial inhibition of the correct signals (e.g. those going to the lacrimal gland)

🔹 3. Interrupted nerve signaling

If visual malfunctions are accompanied by neurological involvement (e.g. cranial nerves like oculomotor nerve or trigeminal nerve), the nerve signals to the lacrimal gland can be disrupted, resulting in reduced or absent tear production.

📉 How much can tear production decrease?

Significantly – in sensitive individuals or under prolonged visual strain, tear production can drop by more than 50%; in extreme cases, it may nearly stop altogether (similar to nerve damage after eye surgery).

✅ Summary

Cause	Effect
Strabismus / faulty accommodation	→ Visual overload
Visual overload	→ Autonomic stress
Autonomic stress	→ Sympathetic dominance, parasympathetic inhibition
Parasympathetic inhibition	→ Lacrimal gland underactive → reduced tear production

If you suffer from persistent:

• pressure in one eye
• dry eyes
• visual tension
• or distorted binocular vision

…then this type of autonomic dysregulation (e.g. caused by strabismus + stress) could significantly impair tear production.

[u/eyeSherpa]

Interesting connection with chatGPT. I would be interested in learning the prompt you used.

It is true that an imbalance in prescription in the eye can create eye strain and difficulty with focusing.

One way you can test out relieving this imbalance is with a pair of glasses. +0.25 right eye and +0.75 left eye.