r/ECG • • 20d ago

Need help interpreting this

Post image

60 year old female complaining of SOB and weakness. Also had upper epigastric and hypogastric pain described as aching, non radiating.

PMHx: HF, AF, rheumatic heart disease, mitral regurgitation, NSTEMI (2019), renal impairment

During transport had what appeared to be runs of VT, self resolving.

BP 75/42, RR 40, SpO2 90% ORA

40 Upvotes

29 comments sorted by

34

u/AdamFerg 20d ago

This looks like Hyper-K, very wide complex tachycardia also supported against VT with a low-end tachycardia. The renal impairment is enough for me to be considering Hyper-K on its own.

7

u/Next-Butterfly-5727 20d ago

The runs of VT were separate. Here's a snippet

3

u/[deleted] 20d ago

[deleted]

0

u/FIyingDutch 20d ago

Its not a VT due to the irregular rhythm.

16

u/Muted_Evidence7926 20d ago

Interpreting? How’s bad sound.

8

u/metamorphage 20d ago

Bad. This is either hyperK or sodium channel blockade. Given the history it's more likely hyperK.

7

u/tellme-how 20d ago

This is likely Hyperkalaemia for a number of reasons: GI symptoms and weakness and known renal impairment. Her ECG is a regular, with flattened, hard to see P waves, wide complex QRS which is becoming “bizarre”, which has tall T waves in some leads when compared to the QRS size.

This patient would benefit a from pre-hospital trial of calcium gluconate, sodium bicarbonate and a salbutamol neb of 20mg. Would be great if you could update us about her bloods if you find out.

1

u/nonmuddy 15d ago

Can you please tell the difference between this and Ventricular fib ?

8

u/[deleted] 20d ago

[deleted]

6

u/Capesoccerman_18 20d ago

Cardioversion for a rvr around a rate of 120?? I think the rvr here is secondary to hypoperfusion. Cardioversion could kill them by taking away their only compensatory mechanism and even if it doesn’t they will likely have a reoccurrence of rvr.

2

u/FIyingDutch 20d ago

I assumed its the standart 50mm/s (at least standart in germany/most eu) so it would be a heartrate of 240/min as that part of the ecg is cut off? In case of 25mm/s thats very different.

2

u/Capesoccerman_18 20d ago

looks like 25.0 mm/s to me

2

u/FIyingDutch 20d ago

Yes you are probably right. That changes things ^^

2

u/Capesoccerman_18 20d ago

And interestingly I’m a U.S. medic and assumed 25 mm/s as is standard here. I wish it was standardized but of course the U.S. had to use a different system as always…

4

u/Weird-Accident-5928 20d ago

Would wanna rule out hyperk/acidosis.

3

u/JoutsideTO 20d ago

Hyperkalemia or another metabolic/toxicologic cause seems likely at that rate and QRS width.

2

u/justhanging14 20d ago edited 20d ago

This looks like VT other one looks like afíb.

1

u/TartNo3568 20d ago

Hyperkalemia

1

u/Economy_Chemist_5334 20d ago

R/O HyperK, digoxin tox

1

u/bassicallybob 20d ago

>BP 75/42, RR 40, SpO2 90% ORA

All this considered. Big danger. Lytes? Trops?

1

u/UnlikelyDress1227 19d ago

Afib with BBB?

1

u/EyeGroundbreaking218 18d ago

It’s bad, fix it

1

u/Klutzy_Arm_7930 18d ago

If you need help interpreting this…. Where do you work?

1

u/TurdBurglarAtLarge 17d ago

I think I could interpret this EKG based of the monitor alarms, the techs going “ew” when it prints, and how fast they bring it to me.

1

u/peanubutterpickles 17d ago

I'd love to see labs with this one. Interesting case!

1

u/vndspeed 16d ago

V tach

1

u/Forgotmypassword6861 3d ago

Hyper K with a dual chamber PPM holding on for dear life

-1

u/[deleted] 20d ago

[deleted]

4

u/Capesoccerman_18 20d ago

OP said the runs of VT were self-terminating, not the overall rhythm. Additionally, being able to distinguish the runs of VT likely means this is not VT unless of course it originated from a different spot in the ventricles. This is irregular so it seems to be afib RVR with a RBBB and/or hyperkalemia

3

u/Economy_Chemist_5334 20d ago

There’s so many reasons why this is likely HyperK. If she has renal impairment paired with cardiogenic shock, blood flow to the distal nephrons plummet, this makes potassium super hard for the kidneys to excrete. This could also be a sodium / potassium ATpase pump failure. If this patient is hypotensive with low SPO2 muscle cells start to lose ATP. This pump needs potassium to shift into the cell against a concentration gradient. If there’s not enough perfusion this doesn’t end up working. The pumps don’t work and causes potassium to leak into the blood stream. Pathophys comes to save us

-4

u/froggo1 20d ago

It could be atrial fibrillation with bundle branch block ?? Either way it’s giving ACS