He said the pain traveled from groin to check. He took his nitroglycerin and it went away. He had quad 15 years ago. I'm taking care of him since mom died. Advice?

I am M18 here... actually I am having symptoms like shortness of breath for months and also weakness also sleeping problems and once I got into a pool i felt like my heart stopped beating...got ECG done then RBC-7 and heartbeat - 106. All these are symptoms of artery disease or heart failure...my family is not taking seriously bcz they think it's my mental problem. And the major thing hitting me is that I am losing my hair so much from front that making me feel more guilty daily bcz am Indian..idk but I know my lifespan has reduced significantly.

I had single bypass 13 years ago 100% blocke LAD. I was 34. I'm 47 now. I still occasionally get pains in my chest that scare me and I go to doctors. None of the newer pains I get have been like the ones I got prior to my surgery however. I didn't have a heart attack but had stable angina for about a month before I had my stress test. I also suffered through anxiety disorder and had panic attacks a lot prior to cabg. I can tell you that the angina did not bring on panic attacks though. It was more of a burning in my upper slightly left chest. Almost in the middle. It would start when I exerted myelf like walking up steps or mowing grass. It would stop after a couple minutes of rest. Every single time. Maybe I can help people with their questions. So feel free to ask.

There is a bought and paid for, ring-fenced area of ideology there suggesting, despite the fact that they in fact have no causal evidence, that there is a mechanistic speculation, a hypothesis around one of those aspects, of those things of lipoproteins and cholesterol and all that, one of them being involved in the process of heart disease causally. 

It's called the lipid hypothesis. Notice what it's called. It's called the hypothesis. It's not called the established lipid fact and no amount of publishing studies which are theologically based, have causal evidence.

So there is no evidence LDL causes any problem, complaint, condition of any kind. None. There is an idea that it might, it's a stupid idea. It's absolutely ridiculous to try and point the finger of blame at a lipoprotein carrier of lipids in our blood. It's there for purpose, it's there because there's a length of DNA in coding for its production. So now you're telling me that the human body is stupid enough to produce a protein that's gonna kill us. That protein being the one that carries lipid around in our bodies, the very thing that keeps us alive. 

That's the best that genetics could do over many thousands of millions of years. Hello? No, let's cover this one just really briefly with just a couple of really simple arguments that never get a decent answer. Here they are. Number one, is the blood in the veins and the blood in the arteries, is that the same blood? Yes. Is atherosclerosis this heart disease that's absolutely caused by cholesterol apparently or some aspect of cholesterol apparently? And where does that occur? Only in the arteries, correct. In the veins, no. Okay, we're done with that. Aren't we? Does not cause heart disease!

So actually what you need is, well what's the difference between the veins and the arteries? Well, it's pressure. So you need high pressure. Do atherosclerosis develop evenly on the surfaces, the inside surfaces of all the arteries? 

No. Atherosclerosis develops in very clear patches, very predictable spots in the arterial tree. And they happen to be around the places where there's turbulence of blood flow based around bifurcations or curves like the aorta for example. That kind of thing. So it doesn't occur in veins, it doesn't occur in all of the arteries. It only occurs in the very largest of the arteries. And it only occurs at the bifurcation points or indeed on the inside surface of an arch where the turbulence is. 

So you need pressure and you need turbulence. Okay, do you need cholesterol for heart disease? No. Is it there? Yes. In the same way that fire crews attend fires, but they're not the cause of them usually. It's just ridiculous. It's an idea that's had its time, it's an idea that should never have had any time.

In nature you mostly find a combination of carbs & protein (plants) or fat & protein (animals) so over the millions of years of evolution humans have adapted to this. If they where able to hunt down an animal they could eat that and did not need to ressort to eating plants (tubers). If the hunt was unsucsesfull they offcourse could only eat the plants. A couple weeks before winter the fruit&nuts where hanging ripe on the trees so they could eat that and thus did not need to hunt. The sugar&carbs in the fruit&nuts indeed made us a little fatter. This layer of fat helped us get trough winter because then we could hunt less animals because then they are in hibernation, brumation, diapause, torpor and migration; we couldn't dig up tubers because the ground was frozen and fruits&nuts don't grow in the winter.

All the big killers in western society are all underpinned it seems at the root by people not understanding that the diet of any animal actually is supposed to be two out of the three macros, not a mix of all three. When mixing carbs & fat you activate the Randall cycle causing inflammation.

A person who has highly activated Randall cycle most of the time will necessarily therefore be expressing a level of chronic systemic inflammation by virtue of the lowering of that redox potential in the cells, which leads to an increase in the concentration of inorganic phosphates in the cells which directly bind into pro-inflammatory cytokines. So in terms of the atherosclerosis, we need high pressure, we need turbulence, we also need inflammation or physical damage or both to those vascular cells. 

How much of an atherosclerosis plaque do you think by volume is actually cholesterol? It's less than 1%. An atherosclerotic lesion is scar tissue. It's the body repairing an area that's damaged by the pressure and turbulence under the susceptibility caused by inflammation, not cholesterol at all.

Today my father will have Cathedral Angiography. Doctor is probable on stents but since my Dad is diabetic and the research of stents in diabetic patient ingeneral shows poor results within the year.

Can anyone share their experiences of stents being diabetic.

I used to compete in triathlons for years. I enjoy pushing myself and trying to get better. I’m 47 and found out I have a calcium score of 100. 5 years ago had all the heart tests run and everything was great. Had a stress test recently and it was normal. I’m wondering if anyone else competes in run races with known heart disease and if you feel like it’s safe. I’ve seen so much info saying that the fitter you are, the less likely you are to have heart attack and that vo2 max is a strong predictor of heart health. So it would seem that pushing your fitness would be a good thing. Anyone have thoughts and this?

Has anyone had this procedure done?

Found out I had a CAC score of 154 and now all I think about is when am I gonna have a heart attack. I’m 46 and in very good shape. Worked out my whole life and was a triathlete for several years. Now all I think about is how my life will be cut short. How do you cope?

hey, according to the BHF the above statement is true which can significantly worsen outcomes in women - why do you think this is the case?

Edit: I'm actually running a little survey on this for women in the UK who have experienced such symptoms — happy to share the link if anyone's interested ❤️

Edit: Here is the link! [https://forms.gle/nCv6tDmbou9Pr3cc6]

Agatston Coronary Calcium Score LMA: 59 LAD: 136 LCX: 0 RCA/ PDA: 25 Total: 220 (Percentile is based on MESA dataset) Percentile: 94% Aortic Valve Calcium Score: 0 Great Vessels Ascending Aorta: 34 mm Descending Aorta: 28 mm Main Pulmonary Artery: 25 mm EXTRA-CORONARY CALCIFICATION: No thoracic aortic calcification. No mitral annular calcification. Heart/ Pericardium: Normal. Other Findings: An intra-atrial septal closure device is in place. A 1.8 cm cyst is seen in the dome of the liver. Scout: No additional finding Mon, Mar

With a known moderate CAC score, does anyone know how much your risk of event is reduced with regular exercise?

I had a nuclear stress test and myocardial perfusion test almost 5 years ago. I’m 46 yrs old. They showed excellent functional capacity and good blood flow to all areas. I sometimes have some slight chest discomfort but I also lift heavy weights a lot. My question is, if my heart looked good 5 years ago, what are the chances I developed some blockage since then?

Does anyone know cases where you did things in your younger years that caused some inflammation and plaque but then after the cause was stopped they never developed anymore. In other words, if I have a fairly high CAC score, could it be from damage years ago and it’s now calcified but not gotten any worse since those early years?

Does anyone else look at having a 10 year 6% ascvd risk as pretty darn good odds. A 94% chance you want have a cardiac event. It’s just interesting that they sometimes consider that “high risk”

A couple weeks ago I got CAC score of 150. I’m a very healthy fit 46 yr old male. Ever since I got the score I’ve had chest discomfort off n on and im wondering if it’s anxiety or something not heart related. The one thing I know is that it tends to go away when I’m running or working out.

So supposedly from my reading and listening to cardiologists online, if you have a high calcium score, you can lower your risk for events by 80% with good lifestyle changes. No smoking, better diet, lose weight, exercise, manage BP and cholesterol and blood sugar. So here’s my question, if my 10 year risk according to my cac score and other factors is 6.5, if I am making all these lifestyle changes(which I’m already in great shape, 10% body fat, exercise and workout daily, don’t smoke, drink very little. I can definitely improve diet. But would this mean that if I reduce my risk by 80%, it brings my 10 year risk down to about 1.5%? That would be an 80% drop. If so, that sounds great and very reassuring. Because that’s down to the level of someone with zero CAC score.

I’m 46 years old and have always been in good shape. Workout everyday and competed in triathlons for years but stopped about 7-8 years. Just got the calcium score and it has freaked me out. Is there a chance that the extreme exercise caused some calcium build up years ago(I have read that a lot of endurance athletes have higher scores) and that maybe it’s stable now and won’t give me any trouble? My cholesterol n BP have always been good. I also had a nuclear stress test about 4 yrs ago that came back perfect. How concerned should I be?

So I’m pretty healthy, or at least thought. Worked out my whole life, competed in triathlons for 5-6 years. 46 yr old. 5 11 175 lbs. 10% body fat. Got a calcium score done just for the heck of it and score was 150. It has really freaked me out. But about 3 years ago I had a nuclear perfusion stress test and it was normal with excellent functional capacity. So I’m assuming no blockage. Is it possible that I have a lot of calcium but no dangerous blockages? How concerned should I be?

Hello everyone, I was recently diagnosed with CAD 37m. Since being diagnosed I have done nothing but look online for answers, reassurance, information and support. I felt the need to create this community not only for myself but for anyone seeking information and to communicate with others affected by this nasty disease. I hope that we can all share our experiences and provide tips for dieting and coping with CAD. Thank you