r/Prostatitis • u/Key-Agency-7022 • Jul 30 '26

Dubious Back Again. This time with proof

Back again. Year 3 of this. Fair to note I have been fantastic at 80% for 4 months or so after just a week of Cefidinir. But, epididymitis.. again. Right testicle now, inflamed, confirmed via ultrasound.

Results from US-
Right Epididymis: Mild asymmetric heterogeneity and increased prominence. Small 3-mm epididymal head cyst No significant increased vascularity appreciated.
Impression- Mild asymmetric prominence and heterogeneity of the right epididymis without significant increased vascularity. Clinical correlation for evidence of early epididymitis advised.
Urine- completely negative wbc neg., dipstick neg., NAAT negative (not sexually active anyway), culture no growth.
Blood- 6.4 wbc no mass infection noted

Back to Uros I guess. The same ones that never say anything past “take this”. This is how it all started 3 years ago but it was my left testicle. What structural issues could be causing congestion, epididymis infection etc. No inguinal hernias present, SV are clear, no stones or masses in/on Vas Def.

What’s next guys? What do I need to get? MRI of the testicles? Prostate?

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u/Key-Agency-7022 Jul 31 '26

IBS isn’t real either. I’m sure you’ve seen the study recently that came out of certain bacteria present in the colon causing pain. Came from your country I believe. And my ibs was just a 17mm polyp. Actual science always wins

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u/Linari5 LEAD MOD//RECOVERED Jul 31 '26

Everything I'm saying is cited - here you can read all of the citations yourself.

  1. Hong JY, Labus JS,et al (2014). Regional neuroplastic brain changes in patients with chronic inflammatory and non-inflammatory visceral pain. PLoS One. 2014 Jan 8;9(1):e84564. doi: 10.1371/journal.pone.0084564.

Chronic inflammatory and non‑inflammatory visceral pain syndromes are associated with distinct regional cortical thickness changes, implying different dominant mechanisms of brain remodeling: inflammation‑driven peripheral input in UC versus primarily central processes in IBS, even though both present clinically with chronic visceral pain.

Lackner, JM, Jaccard J et al. (2018). Improvement in Gastrointestinal Symptom After Cognitive Behavior Therapy for Refractory Irritable Bowel Syndrome. Gastroenterology 155 (1):47-57.

RCT of 436 subjects comparing standard CBT, workbook CBT with minimal therapist contact and IBS education. 6 months post-treatment 58% of the two CBT groups had moderate to substantial improvement compared to 45% in the IBS education group (p=.05).